Super evil. My brother-in-law dropped dead 18 months to the day after his second shot. No boosters after that. Never had covid. Never tested positive for covid. The autopsy showed thrombocytopenia as a probable cause of death for a sudden and unexpected massive bleed in his chest. He was never diagnosed with thrombocytopenia when he was alive. My husband's primary told us the autopsy was incomplete.
There is evidence that the SarsCov-2-Spike-protein accumulates in the bone marrow of the skull, enters the meninges, and then enters the brain. This pathway could lead to significant brain damage if it causes a disruption of the blood-brain barrier. Additionally, there is evidence that lingering Spike protein is found in some individuals who recovered from COVID-19 and died due to non-COVID-related causes. Complement proteins and coagulation factor 11 contribute to the inflammatory response in COVID-19 patients. Disruptions in complement components were observed in non-survivor COVID-19 patients. Neutrophils may move into the meninges and induce net formation, which is associated with cytokine storms in COVID-19 patients. However, there is no known way to predict which patients are at higher risk for cytokine storms.
Super evil. My brother-in-law dropped dead 18 months to the day after his second shot. No boosters after that. Never had covid. Never tested positive for covid. The autopsy showed thrombocytopenia as a probable cause of death for a sudden and unexpected massive bleed in his chest. He was never diagnosed with thrombocytopenia when he was alive. My husband's primary told us the autopsy was incomplete.
There is evidence that the SarsCov-2-Spike-protein accumulates in the bone marrow of the skull, enters the meninges, and then enters the brain. This pathway could lead to significant brain damage if it causes a disruption of the blood-brain barrier. Additionally, there is evidence that lingering Spike protein is found in some individuals who recovered from COVID-19 and died due to non-COVID-related causes. Complement proteins and coagulation factor 11 contribute to the inflammatory response in COVID-19 patients. Disruptions in complement components were observed in non-survivor COVID-19 patients. Neutrophils may move into the meninges and induce net formation, which is associated with cytokine storms in COVID-19 patients. However, there is no known way to predict which patients are at higher risk for cytokine storms.